Ginkgolide C

CAS No. 15291-76-6

Ginkgolide C ( BN 52022; 1,7-dihydroxy Ginkgolide A )

Catalog No. M12143 CAS No. 15291-76-6

Ginkgolide C, a natural product, can enhance the cardiac function of rats in the body.

Purity : >98%(HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
25MG 160 Get Quote
50MG 245 Get Quote
100MG Get Quote Get Quote
200MG Get Quote Get Quote
500MG Get Quote Get Quote
1G Get Quote Get Quote

Biological Information

  • Product Name
    Ginkgolide C
  • Note
    Research use only, not for human use.
  • Brief Description
    Ginkgolide C, a natural product, can enhance the cardiac function of rats in the body.
  • Description
    Ginkgolide C, a natural product, can enhance the cardiac function of rats in the body.
  • Synonyms
    BN 52022; 1,7-dihydroxy Ginkgolide A
  • Pathway
    Others
  • Target
    Other Targets
  • Recptor
    Others
  • Research Area
    Other Indications
  • Indication
    ——

Chemical Information

  • CAS Number
    15291-76-6
  • Formula Weight
    440.40
  • Molecular Formula
    C20H24O11
  • Purity
    >98%(HPLC)
  • Solubility
    DMSO: 10 mM
  • SMILES
    C[C@@H]1C(O[C@H]2[C@@H]([C@]34[C@@H]5OC([C@@]3([C@@]12O)O[C@@H]6OC([C@@H]([C@]46[C@H](C(C)(C)C)[C@H]5O)O)=O)=O)O)=O
  • Chemical Name
    Ginkgolide A, 1,7-dihydroxy-, (1alpha,7beta)-

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1. Liou CJ, et al. Evid Based Complement Alternat Med. 2015;2015:298635.
molnova catalog
related products
  • NSC348884

    NSC348884 is a nucleophosmin inhibitor disrupts oligomer formation and induces apoptosis, inhibits cell proliferation at an IC50 of 1.7-4.0 μM in distinct cancer cell lines.

  • 2-O-Acetyl-20-hydrox...

    2-O-Acetyl-20-hydroxyecdysone, an ecdysterones in insects and terrestrial plants, inhibits amyloid-β42 (Aβ42)-induced cytotoxicity.

  • Retro 2

    Retro 2 is a plant toxin ricin ?inhibitor, itprotects HeLa cells against Ricin, Stx1 and Stx2, selectively blocks retrograde toxin trafficking at the early endosome-TGN (trans-Golgi network) interface.